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余春红 ( 讲师 ) 赞
的个人主页 http://shi.buaa.edu.cn/ych3433/zh_CN/index.htm
讲师博士生导师硕士生导师

论文当前位置：中文主页 >> 论文

[1] .Research progress of artificial blood:Basic & Clinical Medicine
[2] .Proteomics Study of Benzene Metabolite Hydroquinone Induced Hematotoxicity in K562 Cells:Biomedical and Environmental Sciences
[3] .The role of catechol-O-methyltransferase in catechol-enhanced erythroid differentiation of K562 cells:Toxicology and Applied Pharmacology
[4] .Phenolic Metabolites of Benzene Induced Caspase-Dependent Cytotoxicities to K562 Cells Accompanied with Decrease in Cell Surface Sialic Acids:Environmental Toxicology
[5] .Long-term exposure of K562 cells to benzene metabolites inhibited erythroid differentiation and elevated methylation in erythroid specific genes:Toxicology Research
[6] .5-氮杂-2’-脱氧胞苷对苯代谢物抑制K562细胞转铁蛋白受体表达的干预作用:毒理学杂志
[7] .The role of ROS in hydroquinone-induced inhibition of K562 cell erythroid differentiation:Biomedical and Environmental Sciences
[8] .吞蛋白A2在非网格蛋白内吞中的作用研究进展:生物化学与生物物理进展
[9] .Protease sensitivity and redistribution of CD71 and glycophorin A on K562 cells:Cellular and Molecular Biology
[10] .Changes in DNA methylation of erythroid-specific genes in K562 cells exposed to catechol in long term:Toxicology in Vitro

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